Vuez Stang*
Numerous human studies and animal models have pointed to the involvement of various infectious agents in the development of vasculitis, particularly in susceptible individuals. However, the relationship between infection and vasculitis is exceedingly intricate and not yet fully comprehended. What makes it even more complex is that different agents can trigger the same type of vasculitis, as is evident in the case of Leukocytoclastic Vasculitis. Conversely, the same infectious agent can evoke a wide spectrum of host responses. These responses can range from a clinically silent infection or limited involvement of a particular organ to the development of devastating, widespread vasculitis. This variability underscores the multifaceted nature of the interplay between infections and the development of vasculitis in susceptible hosts.
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